I would not make any sudden decisions. Do your research, please request a consultation appointment with your surgeon, and consider additional opinions. You may want to consult with an allergist, or consider a teaching hospital.
I did a quick search for you, and found this interesting article:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3033552/
If this is what you are experiencing, I would investigate the possible treatments:
Antihistamines H2 antihistamines are targeted to decrease hypersecretion of gastric acid and can be helpful for treating the gastrointestinal symptoms of diarrhea and abdominal cramping. Ranitidine 150 mg orally every 12 hours or famotidine 10 mg orally every 12 hours can be used. H1 antihistamines can control flushing and pruritis.26 Patients evaluated in the study by Jakate and coworkers showed significant symptom improvement with H1 and H2 antihistamines.6
Cromolyn An inhibitor of mast-cell degradation, cro-molyn can be used at a dose of 100–200 mg orally 4 times per day. Small studies have shown improvement in gastrointestinal symptoms with cromolyn therapy.26,27 In a large multicenter trial evaluating 428 diarrhea-predominant irritable bowel syndrome patients, oral cromolyn was compared to elimination diet. Symptom improvement was seen in 67% of patients treated with oral cromolynsodium (1,500 mg/day) for 1 month compared to 60% of patients treated with elimination diet.28
Antileukotriene Drugs Although antileukotriene drugs such as montelukast may cause abdominal pain, they have shown improvements in pruritus and flushing.29
Budesonide If the aforementioned, targeted therapies fail, 9 mg daily of oral budesonide can be used as an alternative treatment.16
Exclusion Diet As food allergies have been proposed in the pathogenesis of irritable bowel syndrome, patients with mast-cell infiltration may benefit from allergy testing and exclusion diets.21 The role of non-IgE delayed type 4 reaction is unclear in gastrointestinal food allergies. Diagnosing specific gastrointestinal food allergies is difficult, as IgE immediate type 1 reaction is rare. Methods such as skin prick tests and RASTs for IgE suggest that sensitization to certain allergens may develop. However, a positive test implies a food allergy without a clinical reaction.30,31
A 2-week diet excluding certain foods should be adequate to assess response. Gradual reintroduction of foods accompanied by the development of symptoms suggests the presence of trigger foods. A meta-analysis reviewing 7 studies of elimination diets in patients with irritable bowel syndrome suggested that milk, wheat, eggs, and foods high in salicylates or amines consistently exacerbated symptoms.32 Desensitization injections have been evaluated as a possible treatment, but inadequate evidence exists in IgE-medicated reactions to support their effectiveness.
In conclusion, the authors believe that mast cells may play a common but, heretofore, underappreciated role in gastrointestinal diseases and thus recommend that patients with chronic unexplained diarrhea undergo colo-noscopic biopsies with special mast-cell stains.